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Context, anatomy, and key evidence

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mitochondrial-derived peptide

Humanin

Published evidence · coverage incomplete
Anatomical contextAnatomy connections are not mapped yet.

No body region is highlighted until this profile has a supported anatomical connection. This does not mean the peptide has no biological effects.

Read published mechanism findings ↓

At a glance

What is it—and why does it matter?

Humanin is described as a mitochondrial DNA–encoded peptide (a mitochondria-derived peptide).

Sources for this introduction: [1]

This published profile is a reference in progress. Findings retain their source context; missing topics are marked below.

What is it?

A molecule, not a product name.

Identity

Humanin is classified among mitochondrial-derived peptides, a group of peptides encoded by mitochondrial-origin sequences and discussed in relation to metabolic regulation.

Source records
1
Independent studies
1

Evidence boundary: Applies only to the source-defined population, formulation, dose, and assessment period.

Exact passages, locators, and provenance
  1. supports · Source-backed record
    Mitochondrial-derived peptides (MDPs), such as humanin and MOTS-c, play a role in regulating energy balance and cellular stress responses.

    Mitochondrial-derived peptides (MDPs) activated by physical exercise as therapeutic targets for metabolic disorders: A systematic review. · BACKGROUND

    pubmed:42640735:ffcd622d7fe4:ffcd622d7fe4

Identity

Humanin is described as a mitochondrial DNA–encoded peptide (a mitochondria-derived peptide).

Source records
1
Independent studies
1

Evidence boundary: Applies only to the source-defined population, formulation, dose, and assessment period.

Exact passages, locators, and provenance
  1. supports · Source-backed record
    This study aimed to investigate the role and potential mechanism of the mitochondrial DNA encoded peptide Humanin (HN) in alleviating rotenone-induced neurotoxicity.

    Humanin improved the rotenone-induced reactive oxygen species formation in PC12 cells by modulating the SIRT3/Nrf2/HO-1 signaling pathway. · Abstract

    pubmed:42041115:0151e87542f7:0151e87542f7

How does it work?

Target, response, and disposition.

This profile does not yet contain a published summary for this topic. This is a coverage gap, not evidence that no research exists.

What has been studied?

What the evidence says.

This profile does not yet contain a published summary for this topic. This is a coverage gap, not evidence that no research exists.

Risks and interactions

Risks, organized for scanning.

This profile does not yet contain a published summary for this topic. This is a coverage gap, not evidence that no research exists.

Products and regulatory status

Same ingredient. Different records.

This profile does not yet contain a published summary for this topic. This is a coverage gap, not evidence that no research exists.

Administration context

The practical clinical context.

This profile does not yet contain a published summary for this topic. This is a coverage gap, not evidence that no research exists.

Research status + gaps

What still needs better answers?

  • No publishable claim yet for: administration, contraindication, effect, interaction, mechanism, regulatory, safety
  • 54 compiled claim(s) withheld by automated assurance: assurance_score_below_0.58 (47), assurance_score_below_0.72 (6), current_regulatory_source_required (5), extraction_ambiguity (39), high_risk_requires_regulatory_or_two_independent_sources (6), no_direct_support (52)

These are the limits of this profile, not an exhaustive list of scientific uncertainties.

References + discovery

Open the records yourself.

  1. Humanin improved the rotenone-induced reactive oxygen species formation in PC12 cells by modulating the SIRT3/Nrf2/HO-1 signaling pathway.

    pubmed · published 2026-08-01 · retrieved 2026-08-27T11:48:55Z

  2. Mitochondrial-derived peptides (MDPs) activated by physical exercise as therapeutic targets for metabolic disorders: A systematic review.

    pubmed · published 2026-08-25 · retrieved 2026-08-27T11:48:55Z

Publication history and provenance

Version 3 · Automated assessment · 2026-08-27T11:48:55Z

6e6573c36fb28d8a639b25cc7cf50a6364f9e95b4d87f3bd4911853f3b25fff4