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hunger hormone

Ghrelin

Published evidence · coverage incomplete
Anatomical contextAnatomy connections are not mapped yet.

No body region is highlighted until this profile has a supported anatomical connection. This does not mean the peptide has no biological effects.

Read published mechanism findings ↓

At a glance

What is it—and why does it matter?

In an inositol triphosphate turnover functional assay using [2-3H]myo-inositol in COS 7 cells expressing human ghrelin receptor, CHEMBL500468 shows agonist potency with EC50 = 1.4 nM.

Sources for this introduction: [1]

This published profile is a reference in progress. Findings retain their source context; missing topics are marked below.

What is it?

A molecule, not a product name.

This profile does not yet contain a published summary for this topic. This is a coverage gap, not evidence that no research exists.

How does it work?

Target, response, and disposition.

Mechanism

Using an inositol triphosphate turnover assay with [2-3H]myo-inositol in COS 7 cells expressing human ghrelin receptor, CHEMBL500468 achieves a maximal effect (Emax) of 162.0% relative to untreated control under the assay conditions.

Reported result
Emax = 162.0 %
Source records
1
Independent studies
1

Evidence boundary: Applies only to the source-defined population, formulation, dose, and assessment period.

Exact passages, locators, and provenance
  1. supports · Source-backed record
    Standard result: Emax = 162.0 %

    ChEMBL activities for CHEMBL500468 · Activity 12098317

    chembl-activities:chembl500468:993e87b75c11:993e87b75c11

Mechanism

In a competitive radioligand binding assay using [35S]-MK-0677, CHEMBL500468 inhibits ligand binding to human GHSR1 with an IC50 of 0.25 nM, indicating high-affinity receptor binding under the reported assay conditions.

Reported result
IC50 = 0.25 nM
Source records
1
Independent studies
1

Evidence boundary: Applies only to the source-defined population, formulation, dose, and assessment period.

Exact passages, locators, and provenance
  1. supports · Source-backed record
    Standard result: IC50 = 0.25 nM

    ChEMBL activities for CHEMBL500468 · Activity 439602

    chembl-activities:chembl500468:993e87b75c11:993e87b75c11

Mechanism

A FLIPR-based cell assay in H4 cells expressing the human GHS receptor reports agonist potency for CHEMBL500468 with EC50 = 1.4 nM, reflecting concentration for half-maximal functional response in that readout.

Reported result
EC50 = 1.4 nM
Source records
1
Independent studies
1

Evidence boundary: Applies only to the source-defined population, formulation, dose, and assessment period.

Exact passages, locators, and provenance
  1. supports · Source-backed record
    Standard result: EC50 = 1.4 nM

    ChEMBL activities for CHEMBL500468 · Activity 2163153

    chembl-activities:chembl500468:993e87b75c11:993e87b75c11

Mechanism

In an inositol triphosphate turnover functional assay using [2-3H]myo-inositol in COS 7 cells expressing human ghrelin receptor, CHEMBL500468 shows agonist potency with EC50 = 1.4 nM.

Reported result
EC50 = 1.4 nM
Source records
1
Independent studies
1

Evidence boundary: Applies only to the source-defined population, formulation, dose, and assessment period.

Exact passages, locators, and provenance
  1. supports · Source-backed record
    Standard result: EC50 = 1.4 nM

    ChEMBL activities for CHEMBL500468 · Activity 12098320

    chembl-activities:chembl500468:993e87b75c11:993e87b75c11

Mechanism

In a competitive receptor binding assay (scintillation counting based) using [125I]-His-ghrelin in COS7 cells expressing human ghrelin receptor, CHEMBL500468 shows binding affinity quantified as Ki = 0.7 nM after 75 mins incubation.

Reported result
Ki = 0.7 nM
Source records
1
Independent studies
1

Evidence boundary: Applies only to the source-defined population, formulation, dose, and assessment period.

Exact passages, locators, and provenance
  1. supports · Source-backed record
    Standard result: Ki = 0.7 nM

    ChEMBL activities for CHEMBL500468 · Activity 12098275

    chembl-activities:chembl500468:993e87b75c11:993e87b75c11

Mechanism

In a cell-based radioligand binding assay using [125I][His9]-ghrelin, CHEMBL500468 inhibits radioligand binding to cloned human GHSR type I in LLC PK-1 cells with IC50 = 0.39 nM.

Reported result
IC50 = 0.39 nM
Source records
1
Independent studies
1

Evidence boundary: Applies only to the source-defined population, formulation, dose, and assessment period.

Exact passages, locators, and provenance
  1. supports · Source-backed record
    Standard result: IC50 = 0.39 nM

    ChEMBL activities for CHEMBL500468 · Activity 1142729

    chembl-activities:chembl500468:993e87b75c11:993e87b75c11

What has been studied?

What the evidence says.

This profile does not yet contain a published summary for this topic. This is a coverage gap, not evidence that no research exists.

Risks and interactions

Risks, organized for scanning.

This profile does not yet contain a published summary for this topic. This is a coverage gap, not evidence that no research exists.

Products and regulatory status

Same ingredient. Different records.

This profile does not yet contain a published summary for this topic. This is a coverage gap, not evidence that no research exists.

Administration context

The practical clinical context.

This profile does not yet contain a published summary for this topic. This is a coverage gap, not evidence that no research exists.

Research status + gaps

What still needs better answers?

  • No publishable claim yet for: administration, contraindication, effect, identity, interaction, regulatory, safety
  • 35 compiled claim(s) withheld by automated assurance: assurance_score_below_0.58 (30), assurance_score_below_0.72 (4), current_regulatory_source_required (4), extraction_ambiguity (21), high_risk_requires_regulatory_or_two_independent_sources (4), no_direct_support (33)

These are the limits of this profile, not an exhaustive list of scientific uncertainties.

References + discovery

Open the records yourself.

  1. ChEMBL activities for CHEMBL500468

    chembl-activities · published 2026-08-26 · retrieved 2026-08-26T20:13:52Z

Publication history and provenance

Version 4 · Automated assessment · 2026-08-26T20:13:52Z

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